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Venkatesh, Chaithra N, Parthasarathi K Kulkarni, Siddartha H N, Hanumanthachar Joshi K. A Comprehensive Review on Formulation and Evaluation of Vitamin D3 Emulgel for Enhanced Transdermal Delivery. IJRPAS, 2024; 3(1): 46-63.

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A Comprehensive Review on Formulation and Evaluation of Vitamin D3 Emulgel for Enhanced Transdermal Delivery  

Venkatesh,  Chaithra N *, Parthasarathi K Kulkarni, Siddartha H N, Hanumanthachar Joshi K  

Sarada Vilas college of pharmacy, Mysuru, Karnataka, India.

*Correspondence: chaithranarayan25@gmail.com ; Tel.: (9741766113)

INTRODUCTION

Transdermal drug delivery systems have acquired considerable attention in recent years as a non-invasive and patient-friendly approach to administering therapeutic agents. Topical delivery of the drugs implies priority for their unique feature of by-passing first pass metabolism for local and systemic action. Among the essential nutrients, Vitamin D3 holds a pivotal role in various physiological processes, including bone health, immune function and cellular regulation. However, traditional oral supplementation of Vitamin D3 may present challenges related to patient adherence and bioavailability. In response to these challenges, innovative formulations, such as emulgels have emerged as promising vehicles for enhancing the transdermal delivery of Vitamin D3.[1]

Emulgels exhibit thixotropic properties and are characterized by being grease-free, effortlessly spreadable, easily removable, emollient, non-staining, environmentally friendly, transparent, and cosmetically approved. They also have good cutaneous penetration and long shelf-life. These qualities collectively render emulgels a favorable system for delivering topical medications.

Emulgel represent a hybrid dosage form that combines the advantages of both emulsions and gels. This unique formulation seamlessly transitions from an emulsion state, providing stability and solubilization of lipophilic compounds like Vitamin D3, to a gel upon skin contact, offering enhanced adherence and localized drug delivery. The formulation of Vitamin D3 emulgels involves a careful selection of emulsifiers, gelling agents and permeation enhancers to achieve an optimal balance of stability, viscosity and skin permeability.[2]

This study aims to explore the formulation intricacies and evaluate the performance of Vitamin D3 emulgels in facilitating transdermal absorption. The choice of emulsifiers and gelling agents, along with their concentrations, will be systematically optimized to ensure the stability and efficacy of the emulgel formulation. Physicochemical parameters, including pH, viscosity and drug content will be thoroughly characterized to assess the quality of the emulgels.

Skin permeation tests employing Franz diffusion cells with excised skin samples will give insights into the transdermal delivery capability, in addition to the in vitro release studies undertaken to understand the sustained release profile of Vitamin D3 from the emulgels. The in vivo experiments will further evaluate the effectiveness of the created emulgels in contrast to standard formulations by assessing blood Vitamin D3 concentrations in animal models.[3]

By combining advancements in pharmaceutical formulation and transdermal drug delivery, this research seeks to contribute to the growing body of knowledge on Vitamin D3 supplementation. The outcomes of this study may have significant implications for the development of efficient and patient-friendly approaches to enhance Vitamin D3 bioavailability through topical application, addressing current challenges associated with traditional oral supplementation.

Vitamin D is available in two primary forms: D2 and D3, as well as other active analogs. Ergocalciferol (D2) is sourced from fortified milk, herring, mackerel, tuna, salmon, sardines, eggs, as well as fortified cereals and baked goods. Vitamin D3, otherwise known as cholecalciferol, is photochemically produced upon UV exposure from the precursor sterol 7- dehydrocholesterol, which is present in the epidermis of the skin. Vitamin D3 is an essential nutrient and pro-hormone; itis present in animal products and fortified foods and can be consumed from fish oil, eggs or fish. This article outlines a pilot study investigating the feasibility of delivering vitamin D3 topically. Vitamin D is lipophilic (fat soluble) in nature and can be absorbed through the skin, it is stored in the body's fat cells. [4]                       

Vitamin D3, (cholecalciferol) as it is a fat-soluble vitamin that plays a crucial role in calcium and phosphate metabolism. Its physicochemical properties are important factors to consider when formulating pharmaceuticals, supplements, or topical products. Here are some important physicochemical features of  Vitamin D3. [5, 6]

Chemical structure

Figure 1: Cholecalciferol


Vitamin D3 is a secosteroid, which means it has a steroid structure with a broken ring. Vitamin D3 has a steroid nucleus with a side chain in its chemical structure.

Molecular weight

Vitamin D3 has a molecular weight of around 384.65 grams per mole.

Solubility

Vitamin D3 is lipophilic (fat-soluble) and exhibits very low solubility in water. It dissolves readily in fat and oils. This characteristic influences its bioavailability and absorption in the body.

Melting point

The melting point of vitamin D3 is between 83 and 86 degrees Celsius (181 and 187 degrees Fahrenheit).

Stability

Vitamin D3 is sensitive to light, heat, and air. Exposure to these factors can lead to the degradation of the vitamin. It is often recommended to store vitamin D3-containing products in dark and cool conditions to maintain stability.

Bioavailability

The bioavailability of vitamin D3 is influenced by its solubility and absorption characteristics. It is absorbed in the small intestine and undergoes hydroxylation in the liver and kidneys to become the active form of vitamin D.

Photostability

Vitamin D3 is susceptible to photodegradation when exposed to ultraviolet (UV) light. This property should be considered when formulating products containing vitamin D3, and appropriate packaging may be necessary to protect it from light.

 

 

 

Metabolism

The body converts vitamin D3 into calcitriol (1,25-dihydroxyvitamin D3), the active form of vitamin D that is essential for maintaining calcium and phosphorus homeostasis, through a sequence of hydroxylation events that occur in the liver and kidneys.

The active form of vitamin D3 is calcitriol. Vitamin D3, also known as cholecalciferol, is converted into calcitriol through a series of metabolic processes in the body.

The simplified overview of the conversion process:

Production of Vitamin D3

Vitamin D3 is synthesized in the skin when 7-dehydrocholesterol, a compound present in the skin, is exposed to ultraviolet B (UVB) radiation from the sun.

Conversion in the Liver

Vitamin D3 is then transported to the liver, where it undergoes hydroxylation (addition of a hydroxyl group) by the enzyme 25-hydroxylase. This forms 25-hydroxyvitamin D3, also known as calcidiol.

Conversion in the Kidneys

The next step takes place in the kidneys, where 25-hydroxyvitamin D3 undergoes another hydroxylation, this time by the enzyme 1-alpha-hydroxylase. This final conversion results in the formation of the active form of vitamin D3, which is calcitriol (1,25-dihydroxyvitamin D3).

Calcitriol plays a crucial role in calcium and phosphorus metabolism. It acts as a hormone that regulates the absorption of calcium and phosphate from the gastrointestinal tract and helps maintain proper levels of these minerals in the blood.

It's important to note that while vitamin D3 is obtained from sunlight exposure, dietary sources, or supplements, the conversion into its active form primarily occurs in the liver and kidneys. The active form, calcitriol, then exerts its effects on various tissues and organs throughout the body.

Reasons for Vitamin D3 deficiency[7, 8]

Lack of exposure to sun rays.

Individuals who live in areas with extreme pollution levels or in which towering buildings obstruct the exposure to sun rays as well as who will be applying an excessive amount of sunscreen will be impacted by a lack of vitamin D3.

Impaired vitamin D absorption (due to a medical condition)

The body may also be deficient in some nutrients if a person has gastrointestinal diseases, problems with fat absorption, or lactose intolerance or allergy to milk.

Medications that interrupt the regular functioning of vitamin D in the body

Individuals who have undergone weight loss surgery or those who are using anticonvulsant medications.

 

 

Low dietary intake of vitamin D

If milk is not consumed on a regular basis, body might not be able to sustain the necessary amounts.Vitamin D is the only vitamin that the body actually makes. It is made in the skin and converted to a hormone. Autoimmune skin diseases such rosacea, psoriasis, and eczema develop when vitamin D hormone levels are out of equilibrium.

Irregularities in Melanin

Melanin functions as a natural sunblock, reducing the synthesis of vitamin D3 in the skin. Individuals with a darker complexion or more pigmented skin require a longer duration compared to those with fair skin to generate an equivalent amount of vitamin D3.

Role of Vitamin D3 in maintaining healthy skin[7]

Promotes longer telomeres.

It prevents the premature aging of your skin. Telomeres, the caps of genetic material on the free ends of DNA strands, shortens with increased age. The shortening of telomeres results in DNA instability, a condition that continues to deteriorate until the cell undergoes death.

Acts as endogenous antibiotics.

It is associated with antimicrobial peptides (AMP) production in the keratinocytes (skin cells). It aids skin in overcoming conditions including vitiligo, rosacea, acne, psoriasis, and atopic dermatitis, etc.

Modulates the production of keratinocytes.

Regulates skin cell growth, repair and maintains its metabolism. Both the formation of new blood vessels and the process of wound healing are aided by Vitamin D3. Additionally, it keeps the epidermal barrier intact.

Plays a part in the immune responses of the skin.

It oversees the regulatory mechanisms of skin immunity.

Serves as an antioxidant.

Highly reactive oxygen species, known as free radicals, disrupt lipid structures in cell membranes as they seek electrons. They affect the DNA of the cells in a manner utter damaging. If the same process is repeated every time, the damage bulks up and can lead to different skin cancer forms.

Vitamin D serves as a membrane antioxidant, preventing the degradation of epidermal lipids. It neutralizes the free radicals, rendering them harmless. However, the scavenging capability of vitamin D has not been conclusively demonstrated to a satisfactory extent. Dermatologists advocate for well-conducted, high-quality studies to affirm the antioxidant role of vitamin D.

Skin barrier function

Vitamin D plays a crucial role in controlling the production of keratinocytes, the cells found in the epidermis, the outermost layer of the skin. These cells are essential for preserving the functionality of the skin barrier. In addition to keeping dangerous chemicals and bacteria out of the skin, the skin barrier also aids in retaining moisture, preventing the skin from becoming dehydrated.

Boosting skin immunity

One of the main elements of the body's first line of protection is the skin, which is the greatest defensive organ. Vitamin D is essential for activating immune cells like macrophages and monocytes. These cells, part of the innate immune system, are vital for detecting and combating foreign substances entering the skin. Additionally, Vitamin D is necessary for the regulation of B cells and T cells, the main components of the adaptive immune system. Excessive activation of these cells can contribute to the development of autoimmune disorders.

Antimicrobial effects

Vitamin D has a direct antibacterial impact on skin cells in addition to supporting barrier function and controlling immune system components. Vitamin D triggers specific receptors that destroy bacteria when they enter the skin.

Photoprotective effects

The application of vitamin D3 topically has demonstrated protective effects against UV light-induced skin damage. Vitamin D helps decrease cell mortality, enhance cell survival, and alleviate redness resulting from photodamage caused by UV radiation, to some degree.

Healing wounds

Vitamin D has been demonstrated to elevate the expression of cathelicidin, a protein with antimicrobial properties. This protein plays a crucial role in repairing damaged tissue and restoring proper mechanisms, making it essential for the skin's wound healing process.

Role of vitamin D3 on alleviating symptoms of skin infections.[8]

Psoriasis

Psoriasis refers to a condition where skin cells accumulate, leading to the formation of scales and dry, itchy patches. The objective of vitamin D and its analogs is to control the rate of skin cell proliferation, reducing the overactivity of these cells and restoring them to a normal state.

Calcipotriene, a synthetic analogue of vitamin D, stands as one of the frequently prescribed medications for addressing psoriasis. Notably, it was the pioneering vitamin D3 analogue employed in the treatment of psoriasis. Specifically, calcitriol ointment, an approved formulation, is recommended for managing mild-to-moderate plaque psoriasis in patients aged 18 years or older.

Ichthyosis

The manifestation of the rare disorder is closely linked to severely dry and thickened skin, a condition often correlated with low levels of vitamin D in the body. Ichthyosis, encompassing a group of genetic skin disorders, is distinguished by the presence of dry, scaly skin. While vitamin D3 supplementation is not a primary treatment for ichthyosis, it may have certain benefits for individuals with this condition. It provides anti inflammatory effects, moisturizing and barrier functions.

Eczema (Atopic dermatitis)

There are two primary causes of the itchiness and inflammation associated with this condition: improper function of the skin's epidermal barrier, Dysregulated immune response of the skin, which interacts with the environmental factors.

The topical use of vitamin D3 and its analogs increases the vitamin D receptors (VDR) in the skin cells, which improves the epidermal barrier breakage. This, in turn, lessens the transepidermal water loss there by retains moisture for longer time.

Acne

The studies conducted so far have not established a concrete link between acne and vitamin D. If the acne is primarily due to bacterial overgrowth, vitamin D's anti-microbial properties (when topically used) can calm down the symptoms.

The anti-inflammatory nature of vitamin D may help to address the inflammation and redness during recurrent acne.

Vitiligo

The emergence of this uncommon disorder is intricately connected to the pronounced dryness and thickening of the skin, a condition frequently associated with diminished levels of vitamin D in the body. Ichthyosis, representing a collection of genetic skin disorders, is characterized by the notable presence of dry and scaly skin. "Vitamin D protects the epidermal melanin unit and restores its integrity."

Current application of topical vitaminD3

Vitamin D is present in a variety of over-the-counter (OTC) skincare products, serving as either the primary active ingredient or an augmenting supplement. These products include moisturizers, neck creams, oils, face washes, mist drops, sunscreens, and more. Given that vitamin D dissolves in fats and oils, many topical formulations are oil-based. The primary role of vitamin D in these products is to function as a moisturizer and emollient, creating a thin protective layer on the skin that effectively retains moisture. Also, it helps to soften the skin and revive the dry textures.

Conventional dosage forms of vitamin D3 typically refer to the traditional pharmaceutical formulations or presentations in which vitamin D3 is administered for therapeutic purposes. Some common conventional dosage forms of vitamin D3 include:

Oral Tablets/Capsules: Vitamin D3 is often formulated into oral tablets or capsules, allowing for convenient and standardized dosing. These dosage forms are taken orally and are designed for systemic absorption through the digestive tract.

Liquid Formulations: Vitamin D3 can also be formulated as a liquid, often in the form of drops or solutions. This is especially useful for individuals who may have difficulty swallowing solid dosage forms, such as infants or elderly individuals.

Injectable Formulations: Vitamin D3 injections via intramuscular or intravenous routes are used to treat specific medical conditions, including malabsorption problem, osteoporosis, and chronic kidney disease. Injectable forms are typically used under medical supervision and are reserved for specific conditions where rapid and precise dosing is required.

Topical Preparations: While less common, there are also topical formulations of vitamin D3 available, such as creams or ointments. These are applied directly to the skin and are designed for localized effects.

At present, vitamin D supplements are available as oral and injection forms; however,   compliance of oral vitamin D supplementation alone is only reported to be 20-60%. Consequently, the goal of topical vitamin D therapy is to improve patient compliance.

Formulation considerations[9, 10]

Due to its lipophilic nature, vitamin D3 is often incorporated into formulations containing fats, oils, or other lipids. It may be used in supplements, pharmaceuticals and topical products.

It is essential to comprehend these physicochemical characteristics in order to formulate vitamin D3-containing topical products effectively. Formulators need to consider stability, bioavailability, and compatibility with other ingredients to ensure the efficacy and quality of the final product.

Active Ingredient

Vitamin D3 (cholecalciferol) is the active ingredient. The concentration of vitamin D3 in the emulgel should be determined based on the intended use and therapeutic requirements. The concentration of vitamin D3 in topical formulations can vary depending on the intended use, formulation type, and specific product design. Generally, concentrations of vitamin D3 in topical formulations range from 1,000 to 5,000 international units (IU) per gram. It's important to comply with regulatory guidelines for the allowed concentration of vitamin D3 in cosmetic or pharmaceutical formulations.

Base Ingredients

Water - Forms the aqueous phase of the emulgel. Ex: Rose water, sterile water

Oil Phase - Typically includes oils such as mineral oil, sunflower oil, Coconut oil or other suitable oils.

Emulsifiers - Necessary for stabilizing the emulsion and preventing phase separation. Common emulsifiers include Tween-(20,40,60,80), PEG-(300,400,600), Span-(20,40, 60,80), cetyl alcohol, or glyceryl stearate.

Gelling Agents[11, 12]

Gelling agents play a crucial role in the formulation of emulgels, providing the desired texture, stability and rheological properties to the product and to improve gel like consistency of the formulation. When formulating a vitamin D3 emulgel, the choice of gelling agent is important for achieving the desired consistency and user experience. Some of the common gelling agents that could be used in the formulation of vitamin D3 emulgels.

Carbomers - Carbomers, such as Carbopol, are synthetic polymers that can form clear gels when neutralized in an aqueous environment. They provide a smooth and pleasant texture to emulgels. Carbomers are effective in stabilizing emulsions and can be used in a wide range of pH conditions.

Xanthan Gum - Xanthan gum is a natural polysaccharide produced by the fermentation of sugars by the bacterium Xanthomonas campestris. It is recognized for its ability to thicken and stabilize.

Hydroxyethylcellulose (HEC) – HEC is a water-soluble polymer derived from cellulose. It is often used as a thickening agent in emulgels. HEC provides a smooth and non-greasy texture, improving the spreadability of the emulgel on the skin.

Gellan Gum - Gellan gum is a microbial polysaccharide that forms gels with unique properties. It can be used in combination with other gelling agents to achieve the desired consistency in emulgels. Gellan gum provides a stable gel structure and has good rheological properties.

Bentonite Clay - Bentonite is a natural clay that can be used as a gelling agent in emulgels. It also has the benefit of absorbing excess oil, making it suitable for emulgels with a less greasy feel.

Polyvinyl Alcohol (PVA) -  PVA is a water-soluble synthetic polymer that can be used as a gelling agent in emulgels. It is known for its film-forming properties and can contribute to the stability of the formulation.

The choice of gelling agent depends on various factors, including the desired product characteristics, compatibility with other formulation components, and the intended use of the emulgel. Experimentation and optimization may be necessary to achieve the right balance of texture, stability, and performance in a vitamin D3 emulgel.

Penetration enhancers

A penetration enhancer, also known as absorption enhancer, is a substance that is added to emulgel to improve the penetration of drugs or active ingredients through the skin. It includes Propylene glycol, clove oil, isopropyl myristate, olive oil, urea, DMSO.

Co-Solvents

Co-solvents may be added to enhance solubility and stability. Common co-solvents include propylene glycol or glycerin.

Preservatives

Preservatives are essential to prevent microbial contamination and extend the shelf life of the emulgel. Examples include parabens, phenoxyethanol, or benzalkonium chloride.

Antioxidants

Antioxidants like tocopherol (vitamin E) may be included to protect the formulation from oxidation and improve stability.

pH Adjusters

pH adjusters are used to maintain the emulgel within a suitable pH range for stability and skin compatibility. Citric acid, sodium hydroxide, triethanolamine may be used for pH adjustment.

Fragrance and Colorants

Fragrance and colorants may be added for sensory appeal. It's essential to choose ingredients that are compatible with the formulation and do not cause irritation.

Emollients and Humectants

Emollients, such as fatty alcohols or esters, can contribute to the skin-feel of the emulgel. Humectants like glycerin help maintain skin hydration.

Thickeners

Thickeners may be included to enhance the viscosity of the emulgel. Examples include hydroxyethyl cellulose or carbomer.

Stabilizers

Stabilizers may be added to improve the stability of the emulgel over time. This can include stabilizers for the emulsion system and the active ingredient.

Many Obstacles may encounter while formulating vitamin D3 emulgel

Formulating a vitamin D3 emulgel poses several challenges due to the unique characteristics of both the active ingredient (vitamin D3) and the emulgel formulation. Emulgel is a combination of an emulsion (oil-in-water or water-in-oil) and a gel, providing a stable and easily spreadable form. Here are some challenges in the formulation of vitamin D3 emulgel:

Solubility of Vitamin D3

Vitamin D3 is a fat-soluble vitamin, which means it has low water solubility. Incorporating it into a water-based emulgel can be challenging. Techniques such as using solubilizing agents or selecting appropriate oils with good solubilizing properties may be necessary.

Chemical stability

Vitamin D3 is sensitive to light, heat, and air. Formulating an emulgel that protects vitamin D3 from degradation is crucial. The choice of antioxidants and opaque packaging can help mitigate these stability issues.

 

 

 

Compatibility with emulsion components

A stable emulgel can only be achieved by ensuring compatibility between the emulsion components, such as the water and oil phases. Phase separation brought on by incompatibility may shorten the product's shelf life and efficacy.

Emulsion stability

Emulsions may phase separate over time due to their inherent thermodynamic instability. Creating a steady emulsion that doesn't cream or separate is difficult. The right choice of stabilizing methods, including adding thickening agents, and emulsifying agents is essential.

Skin penetration of vitamin D3

Vitamin D3 needs to penetrate the skin to exert its beneficial effects. Achieving optimal skin penetration can be challenging due to the lipophilic nature of vitamin D3. Enhancers or penetration enhancers may be required to improve skin permeability.

Textures and Aesthetics

Emulgels should have a pleasant texture, be easily spreadable, and provide a non-greasy feel. Achieving the right balance between oil and water phases, as well as selecting suitable gelling agents, is important for achieving the desired texture.

Regulatory compliance

Meeting regulatory criteria for vitamin D3 concentrations, as well as guaranteeing the safety and efficacy of the emulgel formulation, adds another degree of complication. Compliance with health-care standards and regulations is critical for market acceptance.

Consumer Acceptance

The sensory attributes of the emulgel, such as color, odor, and feel, are essential for consumer acceptance. Formulating a product that meets consumer expectations in terms of appearance and usability is crucial for market success. Addressing these challenges necessitates a comprehensive understanding of the physicochemical properties of vitamin D3 and the principles of emulsion and gel formulation. Experimentation and optimization are often necessary to achieve a stable and effective vitamin D3 emulgel.

Steps involved in the formulation[13, 14, 15 ]

Emulgel is formulated by following steps,

a. Selection of components

b. Preparation of emulsion

c. Preparation of emulgel.

a. Screening of components

Drug Solubility was checked in various oils by excess addition of drug followed by continuously stirred for 72 hours to achieve equilibrium. After that samples centrifuged and supernatant was taken and solubility was determined by suitable analytical methods. Then, excipients in each category with the highest solubility of drug are selected for further studies.

Psuedoternary phase diagram:

Surfactant and co-surfactant were mixed in different ratios (2:1, 3:1 and 5:1). Every ratio preferred in increasing amount of surfactant with respect to co surfactant while studying on the phase diagrams. In most of the cases aqueous phase.

(Distilled water) used as dilution media. Oil along with Surfactant and co-surfactant was mixed at different ratios from 9:1 to 1:9 in different vials for its each mixture. Main importance of this is to cover the study which decide boundaries of phases formed in the diagrams. Slow titration of oil and surfactant and co-surfactant is performed and visually observed for transparency of emulsion.

b. Preparation of emulsion: The drug is then solubilized in oil and oil is mixed into mixture of surfactant and co-surfactant, this mixture is then diluted with water to form emulsion of known drug.

c. Preparation of emulgel: Gel base is formulate using 1g of the Carbopol with a required quantity of water and kept for overnight soaking, then prepared emulsion is slowly added with continues stirring. Triethanolamine is added to maintain the pH of formulation. Finally required remaining volume is adjusted by distilled water.

Advantages of vitamin D3 emulgel[16, 17, 18]

Vitamin D3 emulgel formulations offer several advantages, especially when compared to traditional dosage forms. Here are some potential advantages:

Enhanced Skin Absorption

Emulgels are designed to improve the penetration and absorption of active ingredients through the skin. Vitamin D3, when formulated as an emulgel, can potentially be absorbed more efficiently through the skin.

Topical Application

Emulgels allow for the topical application of vitamin D3, which is particularly beneficial for individuals who may have difficulty taking oral supplements. This can be useful for people with gastrointestinal issues or those who prefer non-oral methods of supplementation.

Localized Delivery

Emulgels enable the localized delivery of vitamin D3 to specific areas of the skin. This can be advantageous in cases where targeted therapy is needed, such as for skin conditions or joint pain.

Reduced Systemic Side Effects

By delivering vitamin D3 directly to the skin, the risk of systemic side effects may be reduced compared to oral supplementation. This is because topical application can limit the amount of the vitamin that enters the bloodstream.

Stability and Shelf Life

Emulgels can enhance the stability and shelf life of vitamin D3. The gel matrix can protect the active ingredient from degradation, oxidation, or other factors that may affect its efficacy over time.

Ease of Application

Vitamin D3 emulgels are typically easy to apply and spread evenly on the skin. This ease of application can improve patient compliance, as individuals are more likely to adhere to a treatment regimen that is convenient and user-friendly.

 

Reduced Irritation

The emulgel formulation can provide a smooth and non-irritating application, making it suitable for individuals with sensitive skin. This is particularly important for long-term use.

Customization of Formulation

Emulgels allow for flex19ibility in formulating the product with various additives, such as moisturizers or skin-conditioning agents, to enhance the overall skin feel and provide additional benefits.

Disadvantages of vitamin D3 emulgel [ 19]

While vitamin D3 emulgel formulations offer several advantages, there are also potential disadvantages that should be considered. Here are some possible drawbacks:

Skin Irritation

Some individuals may be sensitive to the components of emulgels, leading to skin irritation. This can be a concern, especially for those with pre-existing skin conditions or allergies.

Potential for Overdose

Topical formulations may pose a risk of overdose if not used as directed. It's important for users to follow the recommended dosage instructions to avoid excessive absorption through the skin.

Limited Absorption

While emulgels are designed to enhance absorption, the skin still presents a barrier that may limit the amount of vitamin D3 absorbed. This could be a concern if high doses are required for therapeutic purposes.

Staining and Residue

Some emulgels may leave a residue on the skin, and certain formulations may have the potential to stain clothing. This can be a cosmetic concern for users.

Complex Formulation

The formulation of emulgels can be complex, requiring careful consideration of ingredients and their interactions. Achieving a stable and effective formulation may be challenging, and variations in the manufacturing process could impact product quality.

Cost

Emulgel formulations may be more expensive to produce compared to traditional oral supplements. This could result in higher costs for consumers.

Not Suitable for Systemic Deficiency

Topical formulations are generally more suitable for localized conditions or situations where targeted therapy is needed. They may not be the best option for individuals with systemic vitamin D deficiency, where oral supplementation may be more appropriate.

Compliance Issues

Some individuals may find it less convenient to apply a topical emulgel regularly, leading to issues with compliance. Oral supplements may be more convenient for those who prefer a once-daily dosage.

Risk of Contamination

Like any topical product, emulgels may be at risk of contamination if proper hygiene and storage practices are not followed. This could compromise the safety of the product.The formulation of a vitamin D3 emulgel involves combining water, oil, emulsifiers, and other ingredients to create a stable and effective product. Here are the key formulation requirements for a vitamin D3 emulgel.

CHARACTERIZATION OF VITAMIN D3 EMULGEL

1. Physical examination[20]

Physical examination like Color, homogeneity, consistency are examined visually.

2. Determination of pH:[21]

Numerous Topical formulations have pH range in between of 5-6 measured by using pH meter. For pH determination, take 1g of product and dissolve in 10ml water. pH 1% solution in water of emulgel subjected to measure pH by the digital pH meter. pH of each formulation is done on triplicate to minimize error.

3. Globules size measurement:

To measure this parameter 1.0 gm of product was dissolved in water and stirred to become dispersion and then sample was inserted into the photocell of Malvern zetasizer.

4. Swelling Index:

1 gm of prepared emulgel is taken on porous aluminum foil which is then dispered in 10 ml of 0.1 N NaOH solutions. Sample removed on various time interval and weight is noted till no further change in weight. It is calculated by using following formula:

Swelling Index (SW) % = [[Wt-Wo]/Wo] × 100

Where,

(SW) % = Percentage swelling,

Wo = Original weight of emulgel,

Wt = Weight of swollen emulgel at time t.

 5. Measurement of Bioadhesive strength:

Accurately 1 gm of emulgel is applied between slides containing rat’s hairless skin pieces. Putting weight on single glass slide create some pressure to removed sandwich of two slides. Adding extra weight is concidered as 200 mg/min to until the detachment of the skin surface. Required weight to detach the emulgel from skin will give bio adhesive strength.

It is calculated by using following formula:

Bio adhesive Strength = W / A

where, W= Weight required (in gms) and A=Area (cm2)

6. Determination of Rheological properties:[23]

20gm of prepared emulgel filled in 25ml beaker was used to measure viscosity by using Spindle number S64 by Brookfield viscometer.

7. Accelerated stability studies:[22]

As given in ICH guidelines, the formulations are kept in oven at 37±2°C, 45±2°C and 60±2°C differently for 3 months. Drug content is examined every two week by appropriate analytical method. Stability measurement is based on change in pH of gel or degradation of drug.

8. Determination of % drug content: [26]

1 g of prepared emulgel is mixed with 25 ml of methanol. This resultant solution is sonicated for 30 min. Drug content was analyzed using the suitable analytical method from this solution.

OR

1 gm of emulgel was dissolved in 50 ml of 0.1N NaOH kept aside for 2 hr. Then 5 ml of sample was withdrawn and absorbance was measured at 265 nm by UV visible spectrophotometer (schimadzu).

9. Determination of emulgel spreadability:[19]

It can be determined by using Slip and Drag method, as suggested by Mutimer, For this take 2gm of emulgel and applied on lower side slide which is mounted with wooden block and sandwiched is prepared by using other glass slide having same size which is bind with hook having 500mg weight placed. After 5 min additional weight was placed on pan which connected with second slide. Time to cover 5cm distance for upper slide was recorded and used to calculate spreadability by using following formula:

Spreadability (S) = M×L / T Where,

M = Weight tied to upper slide,

L = Length of glass slides

T = Time taken to cover distance by upper slide.

10. Skin irritation test:[27]

0.25 gm of prepared emulgel is applied to each different site (two to three sites/rabbit). Rabbit skin sites are washed and wiped after 24 hours of treatment, and any unfavorable morphological changes or changes in skin color are noted.

11. In-vitro Diffusion studies:[21]

Franz diffusion cell is used to demonstrate diffusion study of prepared emulgel. A cellophane membrane is used during the study and 0.5g of sample spread on membrane and diffusion is conducted for 8 Hrs at 37±1°C using phosphate buffer (pH 7.4). At the time interval of 1 Hr. 1 ml sample is collected and replaced with fresh buffer solution. Collected samples are analyzed by using suitable analytical method.

12. Syneresis measurement test:[23]

Syneresis measurement test on rest gel shrinks and little liquid is pressed out called syneresis. This could be measured by means of centrifuge tubes in specific apparatus.16

Syneresis(%) =   x 100

13. Determination of Skin Permeation:[24]

Differential scanning calorimetry is used to examine the structural and chemical alterations in the epidermal layer (DSC). Using the DSC method, temperature changes in rats' desiccated SC membranes are examined in order to evaluate the penetration process. In order to guarantee reducing hydration to 20%, both treated and untreated skin samples were previously hydrated on a 27% Sodium-Br solution for at least 48 hours. Before being analyzed, the skin samples are kept in desiccators for three days at silica gel. The skin layer is divided into pieces, and 4 mg of weighted pieces are sealed in 10μL aluminum pans before being added to an empty pan for comparison and put in the differential scanning calorimetry equipment. Nitrogen flow is set to 20 milliliters per minute to act as purge gas. Samples are heated steadily at a rate of 10°C per minute between 30 and 400°C, and any variations in the DSC graph are recorded and examined.

14. Extrudability test (Tube test):[25]

Determines force necessary for removal of emulgel from tube and necessary to evaluate emulgel formulation for extrudability.

15. Microbial assay of emulgel: [27]

Ditch plate technique could be preferred for microbial assay, it is used for evaluation of bacteriostatic or fungistatic activity of an antimicrobial agent and mainly used for semisolid formulation. Zone of inhibition is calculated as per the following equation.

% Inhibition =   × 100

CONCLUSION

In summary, this review emphasizes formulation and thorough evaluation of the vitamin D3 emulgel will present a promising approach for topical application. The optimized emulgel will  exhibits favourable characteristics, including enhanced stability, efficient skin permeation and controlled release of vitamin D3. These findings underscore the potential of the emulgel as a viable and patient-friendly option for delivering vitamin D3 through the skin. However, further investigations, including clinical trials are imperative to confirm its efficacy and safety, paving the way for its eventual incorporation into dermatological practice.

REFERENCES

1.      Ashara K, Soniwala M, Shah K. Emulgel: A novel drug delivery system. Journal of Pakistan Association of Dermatologists. 2016;26(3):244-9.

2.      Peneva P, Andonova V, Pilicheva B, Kassarova M. In vitro survey of Ketoprofen release from emulgels. Medicine. 2014;4(1):118-21.

3.      Khullar R, Kumar D, Seth N, Saini S. Formulation and evaluation of mefenamic acid emulgel for topical delivery. Saudi pharmaceutical journal. 2012 Jan 1;20(1):63-7.

4.      Mandal S, Mandal SS, Sawant KK. Design and development of microemulsion drug delivery system of atorvastatin and study its intestinal permeability in rats. International Journal of Drug Delivery. 2010 Jan 1;2(1).

5.      Joshi B, Singh G, Rana AC, Saini S, Singla V. Emulgel: a comprehensive review on the recent advances in topical drug delivery. Int Res J Pharm. 2011;2(11):66-70.

6.      Bikle DD. Vitamin D metabolism, mechanism of action, and clinical applications. Chemistry & biology. 2014 Mar 20;21(3):319-29.

7.      Kim GK. The rationale behind topical vitamin d analogs in the treatment of psoriasis: where does topical calcitriol fit in?. The Journal of clinical and aesthetic dermatology. 2010 Aug;3(8):46.

8.      Vieth R, Chan PC, MacFarlane GD. Efficacy and safety of vitamin D3 intake exceeding the lowest observed adverse effect level. The American journal of clinical nutrition. 2001 Feb 1;73(2):288-94.

9.      Shankar R, Tiwari V, Mishra C, Singh C, Sharma D, Jaiswal S. Formulation and evaluation of ketoconazole nanoemulsion gel for topical delivery. American Journal of Pharmtech Reseatch. 2015;5(5):446-62.

10.   Modi JD, Patel JK. Nanoemulsion-based gel formulation of aceclofenac for topical delivery. International Journal of Pharmacy and Pharmaceutical Science Research. 2011;1(1):6-12.

11.   Sailaja AK, Supraja R. An overall review on topical preparation-gel. Innovat International Journal Of Medical & Pharmaceutical Sciences. 2016 Nov;1(1):17-2.

12.   Cevc G, Schätzlein A, Blume G. Transdermal drug carriers: basic properties, optimization and transfer efficiency in the case of epicutaneously applied peptides. Journal of Controlled Release. 1995 Sep 1;36(1-2):3-16.

13.   Shankar R, Tiwari V, Mishra C, Singh C, Sharma D, Jaiswal S. Formulation and evaluation of ketoconazole nanoemulsion gel for topical delivery. American Journal of Pharmtech Reseatch. 2015;5(5):446-62.

14.   Modi JD, Patel JK. Nanoemulsion-based gel formulation of aceclofenac for topical delivery. International Journal of Pharmacy and Pharmaceutical Science Research. 2011;1(1):6-12.

15.   Singh RP, Parpani S, Narke R, Chavan R. Emulgel: A recent approach for topical drug delivery system. Asian Journal of Pharmaceutical Research and Development. 2014 Mar 1:112-23.

16.   Vats S, Saxena C, Easwari TS, Shukla VK. Emulsion based gel technique: Novel approach for enhancing topical drug delivery of hydrophobic drugs. International Journal for Pharmaceutical Research Scholars. 2014;3(2):649-60.

17.   Alexander A, Khichariya A, Gupta S, Patel RJ, Giri TK, Tripathi DK. Recent expansions in an emergent novel drug delivery technology: Emulgel. Journal of Controlled Release. 2013 Oct 28;171(2):122-32.

18.   Bhowmik D. Recent advances in novel topical drug delivery system. The Pharma Innovation. 2012 Nov 1;1(9).

19.   Ashara KC, Paun JS, Soniwala MM, Chavada JR, Mori NM. Micro-emulsion based emulgel: a novel topical drug delivery system. Asian pacific journal of tropical disease. 2014 Jan 1;4:S27-32.

20.   Modi JD, Patel JK. Nanoemulsion-based gel formulation of aceclofenac for topical delivery. International Journal of Pharmacy and Pharmaceutical Science Research. 2011;1(1):6-12.

21.   Sumithapaul, Abhineshkumar, Pramod Yadurkar, Kruthikasawant. Design and Development of multiple emulsions for the enhancement of oral bioavailability of Acyclovir. Drug development and Industrial pharmacy, 39(11), 2013, 1808-1817.

22.   Bonacucina G, Cespi M, Palmieri GF. Characterization and stability of emulsion gels based on acrylamide/sodium acryloyldimethyl taurate copolymer. Aaps Pharmscitech. 2009 Jun;10:368-75.

23.   Mandal S, Mandal SS. Research paper microemulsion drug delivery system: a platform for improving dissolution rate of poorly water soluble drug. Int J Pharm Sci Nanotech. 2011;3(4):1214-9.

24.   Shakeel F, Baboota S, Ahuja A, Ali J, Shafiq S. Skin permeation mechanism of aceclofenac using novel nanoemulsion formulation. Die Pharmazie-An international journal of pharmaceutical sciences. 2008 Aug 1;63(8):580-4.

25.   Charoenrein S, Tatirat O, Rengsutthi K, Thongngam M. Effect of konjac glucomannan on syneresis, textural properties and the microstructure of frozen rice starch gels. Carbohydr Polym. 2011;83: 291-6.

26.   Singla V, Sanini S, Rana AC, Singh G. Development and evaluation of topical emulgel of lornoxicam using different polymer bases. Int Pharm Sci. 2012;2(3):36-44.

27.   Murty SN, Hiremath SRR, Physical and chemical enhancers in transdermal delivery of terbutaline sulphate, AAPS Pharm. Sci. Tech 2001; 2:1–5.



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